Discuss the risk factors for, the pathogenesis of, and the consequences of, coronary artery atherosclerosis and comment on the possible causes and the subsequent pathologic findings that could occur with a total serum cholesterol of 268 mg/dl.
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Discuss the risk factors for, the pathogenesis of, and the consequences of, coronary
artery atherosclerosis and comment on the possible causes and the subsequent
pathologic findings that could occur with a total serum cholesterol of 268 mg/dl.
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Solved in 2 steps
- Select two definitions of a pathophysiology of the acute disease state angina. Select all that apply: O The result of decreased blood flow to the myocardium because of coronary artery spasm or temporary constriction Pounding feeling in the head or chest Dull pain progressing to sharp pain in the lower abdomen O Decreased perfusion to the myocardium results in chest pain Nausea and loss of appetite, with swelling in the legs Select two definitions of a pathophysiology of the acute disease state angina.Discuss the pathophysiology of the clotting cascadeThe pathophysiology of a DVT (Deep Vein Thrombosis) and how it might result in a PE (Pulmonary Embolism) are explained?
- Discuss the modifiable factors of pericarditisDiscuss the general pathology of atherosclerosis, including clinical consequences, and evaluate the modifiable and non-modifiable risk factors for the disease. Provide and diagram/illustartion.Discuss the pathology and risk factors for patients who develop venous thrombosis.?
- Name and explain the mechanism of action of the anticoagulants: calcium chelators (oxalate, citrate, EDTA, fluoride), warfarin, and heparin.Distinguish between arterial (white) and venous (red) clots in terms of composition and sites of formation. Know which anticoagulants would be used to treat each.List 3 commonly used medications related to the concept of Perfusion. How are they related. What assessments are required to safely administer these medications? What are the pertinent nursing considerations?Discuss the role of inflammation in the pathophysiology of cardiovascular diseases, including atherosclerosis, myocardial infarction, and heart failure, and explain how inflammatory processes contribute to disease progression.
