Dylan Mortimer an artist struggled with Cystic fibrosis a disease causes the body to produce thick mucus. In his lungs and digestive system. At genetic level, which of the following is the cause of Cystic fibrosis? * a. Distortion of cells b. Mutation in single gene c. Increase in number of p53 cells d. Insufficient number of nitrogenous bases

Dylan Mortimer an artist struggled with Cystic fibrosis a disease causes the body to produce thick mucus. In his lungs and digestive system. At genetic level, which of the following is the cause of Cystic fibrosis? * a. Distortion of cells b. Mutation in single gene c. Increase in number of p53 cells d. Insufficient number of nitrogenous bases

Biology: The Dynamic Science (MindTap Course List)

4th Edition

ISBN:9781305389892

Author:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Publisher:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Chapter19: Genomes And Proteomes

Section: Chapter Questions

Problem 2TYK: How do pseudogenes differ from genes? a. They are not transcribed. b. They contain longer open...

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How do pseudogenes differ from genes? a. They are not transcribed. b. They contain longer open reading frames (ORFs). c. They do not have introns. d. They use a different genetic code. e. Their protein-coding sequence contains more than one startcodon.
Figure 10.14 Human papillomavirus can cause cervical cancer. The virus encodes E6, a protein that binds p53. Based on this fact and what you know about p53, what effect do you think E6 binding has on p53 activity? E6 activates p53 E6 inactivates p53 E6 mutates p53 E6 binding marks p53 for degradation
Which of the following is not true of somatic cell gene therapy? a. White blood cells can be used. b. Somatic cells are cultured, and the desired DNA is introduced into them. c. Cells with the introduced DNA are returned to the body. d. The technique is still very experimental. e. The inserted genes are passed to the offspring.
1. A monogenic disease is a disease caused by a mutation in a single gene. For instance, sickle-cell anemia is caused by a mutation in the HBB gene, which codes for the B- globin chain of hemoglobin. The beginning of HBB is shown here: 5'-ATGGTGCACCTGACTCCTGAGGAGAAGTCTGCCGTTACT...-3' A. Translate this HBB sequence into an amino acid sequence. B. In terms of amino acids, what is the result of the sickle cell mutation, wherein the bolded red A is changed to a T? This single mutation causes hemoglobin to aggregate, causing red blood cells to deform into a sickle-like shape rather than the normal “biconcave disk" shape. C. What would happen if the bolded blue A were mutated to at T? (This is hypothetical; it's not a mutation found in sickle-cell disease.)
1. Which type of mutation results in no change in amino acid sequence for the protein? a Silent b Missense c Nonsense d Frameshift
2. Consider the scenario where a DNA mutation occurs in a person's skin cells, where the gene p53 becomes damaged, and can no longer be transcribed and translated into a functioning 53kd protein. Why might a mutation of this nature be especially concerning to someone who experiences regular exposure to skin carcinogens, such as UV light? ✓✓✓
7. Scientists studying the genetics of a congenital discase analyzed the chromosomes of the patient and found a large portion of chromosome twenty-three in 188 Chapter 3. Gene expression and protein synthesis chromosome one, and a small part of chromosome one in chromosome twenty- three. What type of mutation is this? To answer the question, please explain: 1) what the mutation is? 2) what types of mutation do you know?
12.What is field of cancerization? Explain part A and B of this figure. How does it explain field of cancerization?| (A) mutation mutation mutation mutation morphologically normal morphologically abnormal, hyperplastic, dysplastic, or neoplastic (D) tumor 1 tumor 2 (8) 7,8,9 10 wild-type p53 only G-T45. wt p53 NORMAL PATCH FIELD CARCINOMA 17p 3p, 9p, 8p, 189 11g Figure 11.14 The Biology of Cancer (0 Garland Sdener 2014)
4. What is sickle cell anemia and what causes it. 5. What is nondisjunction? How does nondisjunction cause disorders? 6. What is Gene translocation? 7. Would a mutation in one of your muscle cells affect your offspring? Why or why not? 8. What 3 things can individuals do to protect themselves from developing mutations?
1. Below is the base sequence of protein for normal hemoglobin and the base sequence for the sickle cell hemoglobin:Normal: GGG CTT CTT TTTSickle: GGG CAT CTT TTTa. Transcribe and translate the normal and sickle cell DNA. b. Identify this as a point or frameshift mutation. Explain. c. If the base sequence read GGG CTT CTT AAA instead, would this result in sickle cell hemoglobin? Explain.
Chapter 6. The organization and expression of lymphocyte receptor genes
1. "What genetic material was used in this research?" 2. How does the completion of this research advance the treatment of cancer? in typed please