mass with calcification and an ipsilateral hypoplastic but functional left kidney. There was neither ascites nor significant abdominal lymph nodes. The conclusion was a suprarenal tumour-adenoma or adrenocortical carcinoma to exclude tuberculous adrenalitis. A CT brain scan was normal. In view of the CT abdominal findings suggestive of adrenal tuberculosis, and ESR 58mm/hr, she was commenced on anti-tuberculous drugs. Ten days after admission, she had nausea, vomiting, fever with chills and extreme lethargy. Cardiovascular examination showed a tachycardia, low volume pulse, BP 70/40 mmHg and blood film showed trophozoites of plasmodium falciparum. Blood cultures done thrice revealed no growth. She was managed with intravenous fluids (dextrose in saline), antimalarials, antibiotics and hydrocortisone. She made remarkable recovery and was maintained on oral prednisolone and fludrocortisone. Create a Nursing Care Plan focusing on the possible adverse effect of administering specific medication based on the case of the patient. You have review and read first the medication intended for this patient before you can proceed to the Nursing Care Plan.

Phlebotomy Essentials
6th Edition
ISBN:9781451194524
Author:Ruth McCall, Cathee M. Tankersley MT(ASCP)
Publisher:Ruth McCall, Cathee M. Tankersley MT(ASCP)
Chapter1: Phlebotomy: Past And Present And The Healthcare Setting
Section: Chapter Questions
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A computerised tomography scan (CT) of the abdomen
showed a non-enhancing oval shaped left suprarenal
mass with calcification and an ipsilateral hypoplastic but
functional left kidney. There was neither ascites nor
significant abdominal lymph nodes. The conclusion was a
suprarenal tumour-adenoma or adrenocortical carcinoma
to exclude tuberculous adrenalitis. A CT brain scan was
normal. In view of the CT abdominal findings suggestive of
adrenal tuberculosis, and ESR
commenced on anti-tuberculous drugs. Ten days after
admission, she had nausea, vomiting, fever with chills and
extreme lethargy. Cardiovascular examination showed a
tachycardia, low volume pulse, BP 70/40 mmHg and blood
film showed trophozoites of plasmodium falciparum. Blood
cultures done thrice revealed no growth. She was
managed with intravenous fluids (dextrose in saline),
antimalarials, antibiotics and hydrocortisone. She made
remarkable recovery and was maintained on oral
prednisolone and fludrocortisone.
58mm/hr, she was
Create a Nursing Care Plan focusing on the possible
adverse effect of administering specific medication based
on the case of the patient. You have review and read first
the medication intended for this patient before you can
proceed to the Nursing Care Plan.
Transcribed Image Text:A computerised tomography scan (CT) of the abdomen showed a non-enhancing oval shaped left suprarenal mass with calcification and an ipsilateral hypoplastic but functional left kidney. There was neither ascites nor significant abdominal lymph nodes. The conclusion was a suprarenal tumour-adenoma or adrenocortical carcinoma to exclude tuberculous adrenalitis. A CT brain scan was normal. In view of the CT abdominal findings suggestive of adrenal tuberculosis, and ESR commenced on anti-tuberculous drugs. Ten days after admission, she had nausea, vomiting, fever with chills and extreme lethargy. Cardiovascular examination showed a tachycardia, low volume pulse, BP 70/40 mmHg and blood film showed trophozoites of plasmodium falciparum. Blood cultures done thrice revealed no growth. She was managed with intravenous fluids (dextrose in saline), antimalarials, antibiotics and hydrocortisone. She made remarkable recovery and was maintained on oral prednisolone and fludrocortisone. 58mm/hr, she was Create a Nursing Care Plan focusing on the possible adverse effect of administering specific medication based on the case of the patient. You have review and read first the medication intended for this patient before you can proceed to the Nursing Care Plan.
A 63-year-ol woman presented with increasing darkening
of the skin, dizziness, and easy fatigability, nausea with
occasional vomiting and progressive weight loss over
eight months prior to presentation. There were no
headaches, blurred
consciousness nor change in her bowel habit. The medical
history and systemic review revealed no abnormality and
were not significant as to the likely cause of her disease
state. Physical examination revealed an elderly lady, pale,
asthenic with generalized hyperpigmentation especially on
the face, oral mucosa, palmar creases and knuckles. No
features of malnutrition or hypovitaminosis.
vision,
and
neither
loss
of
There was no significant peripheral lymphadenopathy.
Main findings in the systemic examination were a pulse of
106 bpm, regular and small; blood pressure 100/60 mmHg
supine and 70/40mmHg sitting. She could not stand on
account of severe postural dizziness. The apex beat was
normal. Fundoscopy revealed a normal fundus. All other
systems were essentially normal. A clinical assessment of
Addison's disease to exclude paraneoplastic syndrome
was made. Laboratory investigations and results are
shown
in
Table
1.
Of note
are
the
anaemia
(haemoglobin10gm/dl), with normal red cell morphology;
ESR 58mm/hr (Westergreen method); fasting blood sugar
was 76mg% and total serum protein of 7.8g/L (albumin-
3.4g/L and globulin 4.4g/L). Plasma cortisol
undetectable at 0
was
and 30 minutes of cosyntropin
administration (0.25 mg). Plasma rennin and aldosterone
activity could not be estimated. HIV screening was
negative (HIV 1 & II) Radiological diagnostic tests included
an abdominal ultrasound, which was reported as showing
normal liver, spleen, pancreas and pelvic organs.
However, the left kidney was not outlined.
A computerised tomography scan (CT) of the abdomen
showed a non-enhancing oval shaped left suprarenal
mass with calcification and an ipsilateral hypoplastic but
functional left kidney. There was neither ascites nor
significant abdominal lymph nodes. The conclusion was a
suprarenal tumour-adenoma or adrenocortical carcinoma
to exclude tuberculous adrenalitis. A CT brain scan was
normal. In view of the CT abdominal findings suggestive of
adrenal tuberculosis, and ESR 58mm/hr, she
commenced on anti-tuberculous drugs. Ten days after
admission, she had nausea, vomiting, fever with chills and
extreme lethargy. Cardiovascular examination showed a
tachycardia, low volume pulse, BP 70/40 mmHg and blood
film showed trophozoites of plasmodium falciparum. Blood
cultures done thrice revealed no growth. She was
managed with intravenous fluids (dextrose. in saline).
was
Transcribed Image Text:A 63-year-ol woman presented with increasing darkening of the skin, dizziness, and easy fatigability, nausea with occasional vomiting and progressive weight loss over eight months prior to presentation. There were no headaches, blurred consciousness nor change in her bowel habit. The medical history and systemic review revealed no abnormality and were not significant as to the likely cause of her disease state. Physical examination revealed an elderly lady, pale, asthenic with generalized hyperpigmentation especially on the face, oral mucosa, palmar creases and knuckles. No features of malnutrition or hypovitaminosis. vision, and neither loss of There was no significant peripheral lymphadenopathy. Main findings in the systemic examination were a pulse of 106 bpm, regular and small; blood pressure 100/60 mmHg supine and 70/40mmHg sitting. She could not stand on account of severe postural dizziness. The apex beat was normal. Fundoscopy revealed a normal fundus. All other systems were essentially normal. A clinical assessment of Addison's disease to exclude paraneoplastic syndrome was made. Laboratory investigations and results are shown in Table 1. Of note are the anaemia (haemoglobin10gm/dl), with normal red cell morphology; ESR 58mm/hr (Westergreen method); fasting blood sugar was 76mg% and total serum protein of 7.8g/L (albumin- 3.4g/L and globulin 4.4g/L). Plasma cortisol undetectable at 0 was and 30 minutes of cosyntropin administration (0.25 mg). Plasma rennin and aldosterone activity could not be estimated. HIV screening was negative (HIV 1 & II) Radiological diagnostic tests included an abdominal ultrasound, which was reported as showing normal liver, spleen, pancreas and pelvic organs. However, the left kidney was not outlined. A computerised tomography scan (CT) of the abdomen showed a non-enhancing oval shaped left suprarenal mass with calcification and an ipsilateral hypoplastic but functional left kidney. There was neither ascites nor significant abdominal lymph nodes. The conclusion was a suprarenal tumour-adenoma or adrenocortical carcinoma to exclude tuberculous adrenalitis. A CT brain scan was normal. In view of the CT abdominal findings suggestive of adrenal tuberculosis, and ESR 58mm/hr, she commenced on anti-tuberculous drugs. Ten days after admission, she had nausea, vomiting, fever with chills and extreme lethargy. Cardiovascular examination showed a tachycardia, low volume pulse, BP 70/40 mmHg and blood film showed trophozoites of plasmodium falciparum. Blood cultures done thrice revealed no growth. She was managed with intravenous fluids (dextrose. in saline). was
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