You have 5 different newly discovered compounds and you want to know if these compounds can possibly cause cancer in humans using AMES test. How do you go about the experiment? Outline the general procedure you will perform and include in your answers the expected result if 3 out of the 5 compounds are potential
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- Mr. Yeboah, diagnosed with a malignant tumour of the liver had it removed and was given a course of chemotherapy. Initially, tumour marker (AFP) activity activity was 7500KU/L (which is very high) but after treatment, this gradually declined to only 5KU/L. A routine follow up test was perfomedafter 3 months and the results was 15KU/L for AFP. The doctor suspected a relapse of the tumour and so referred Mr. Yeboahto an oncologist at a cancer centre who also did a re-check. AFP was recorded to be 5KU/L. Enquiries revealed that the hospital and the cancer centre use different instruments for the measurement of AFP. Both results are normal even though the values are significantly different because different methods were used.a) How can both labs confirm that the results are not clinically significant? b) How can both labs avoid this happening again?A local community is experiencing a surge in lung cancer cases. Residents blame the increase on a chemical plant that opened last year.A number of pollutants emitted from the plant are found to be positive in the Ames Test. What are arguments against this plant being a cause of increased cancer incidences (limit 5-6 sentences)?Discuss why cancer treatment may involve a combinationof chemotherapy, radiation therapy, and surgery.
- D Try Again Your answer is wrong. Please read the Explanation for more help. The patients described in the table below just checked in to the Alex Py Memorial Hospital & Regional Medical Center. Which are headed to the Department of Nuclear Medicine for a radioimaging procedure? Which are headed to the same place for a radiotherapy procedure? patient Ravi is being treated for early-stage prostate cancer. After surgery to remove the tumor, his oncologist recommended 40 Gy of external X-ray radiation, split into 22 doses, to kill any remaining cancer cells. He is here for his 22nd and final treatment. Karen is taking part in a research study aimed at determining whether X-ray irradiation can ease the symptoms of advanced arthritis. Today she is scheduled to receive the first of ten 1 Gy doses of X-rays to her hip and shoulder joints. 12-year-old Isabel is being treated for acute lymphoblastic leukemia (a type of blood cancer), and her oncologist has recommended a round of CAR T-cell…How can I study a protein cancer cells?Why Embryonic stem cells research is the most controversial topic in Medical research in this country today?
- https://youtu.be/w7aIxiZQ60g Multiplexing agglutination https://youtu.be/uWStmyJ5Qc0 This is the multiplexing agglutination. Lab report I don’t really know what to talk about, the data, conclusions and the purpose of this. Need help pleasehi, can I please get help on a case study on nueroanatomy I have been struggling for a couple of hours now and can't seem to understand the study to answer the following questions. is there any way or format that i can get help. I would really appreciate it. thanks! 1. Based on the information in the case, what is the most likely neuroanatomic location for a single lesion that can explain all of the patient’s symptoms and signs? In your own words, explain how you arrived at that localization. 2.What are some possibilities for the nature of the lesion (e.g., stroke, tumor, trauma, etc.)? In your own words, explain your rationale for these options. 3. How does the laboratory data and neuroimaging demonstrate the actual lesion for the patient? Describe how you interpret the data in your own words. 4.How was the patient was treated, and how did they subsequently fare? Describe the treatment plan in your own words.There are three general categories of effects resulting from exposure to low doses of radiation. Theseare: 1. Genetic - The effect is suffered by the offspring of the individual exposed. 2. Somatic - The effect is primarily suffered by the individual exposed. Since cancer is theprimary result, it is sometimes called the Carcinogenic Effect. 3. In-utero - Some mistakenly consider this to be a genetic consequence of radiation exposure,because the effect, suffered by a developing embryo/fetus, is seen after birth.However, this is actually a special case of the somatic effect, since the embryo/fetusis the one exposed to the radiation. State the medical case occured from Chernobyl, Three Mile Island, Fukushima Daiichi nuclear power plant accidents and explain briefly the three above categories in each accidents including the diagnosis and treament (if applicable).
- Question 1. Describe and explain the epidemiological evidence supporting the view that cancer develops through a multi-step process involving increasingly severe stages.. Question 2. Describe and explain the genetic evidence supporting the view that cancer develops through a multi-step process involving increasingly severe stages.This is homework not a test! From NTSA case study https://static.nsta.org/case_study_docs/case_studies/cystic_fibrosis.pdf Please help with questions 2, 3 and 4 of part four 2. "The successful use of gene therapy to cure SCID syndrome (2000) is hoped to be a permanent cure for those patients because a good copy of the problem gene was inserted into the patients' blood stem cells in the bone marrow (hematopoietic stem cells). Once white blood cells enter the blood stream they have a limited life span, on the order of a few week to months. The blood stem cells are the cells that create more white blood cells to replace those that are lost. If the gene was only inserted into the circulating mature white blood cells, the patient would only be temporarily cured until those cells were used up or died." The current gene therapy approaches to cure CF involve inserting a functional CFTR gene into the mature epithelial cells of the lungs. In light of the preceding paragraph, do you think that…Skin cancer carries a lifetime risk nearly equal to that of allother cancers combined. Following is a graph [modified fromK. H. Kraemer (1997). Proc. Natl. Acad. Sci. (USA) 94:11–14]depicting the age of onset of skin cancers in patients with orwithout XP, where the cumulative percentage of skin cancer is plotted against age. The non-XP curve is based on 29,757 cancerssurveyed by the National Cancer Institute, and the curverepresenting those with XP is based on 63 skin cancers from theXeroderma Pigmentosum Registry.