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- It has been suggested that a high-sugar diet may lead to non-alcoholic fatty liver disease. Explain?8. The adipose tissue is not only store triacylglycerols but also is active endocrine organ. Explain this function of the adipose tissuc. For that: a) explain the «clinical obesity» and possible consequences of the discase; b) name the biologically active molecules secreted by the adipose tissue; c) explain how secretion of these molecules changes with development of obesity. 9. A daily dict ofa 55-ycar-old woman, consisted ofa 500 g of carbohydrates, 100g of animal fats and 150 g of proteins, at the background of low physical activity.Adults engaged in strenuous physical activity require an intake of about 160 g of carbohydrate daily but only about 20 mg of niacin for optimal nutrition. Given the role of niacin in glycolysis, how do you explain the observation?
- Patient who had undergone a complex maxillofacial operation could not takefood during 2 days, but glucose level in the patient's blood during that period wason the lower limit of normal. What mechanisms facilitate the maintenance ofnormal level of glucose in prolonged starvation? To answer this question:a) indicate normal fasting blood glucose level;b) draw the metabolic pathway ensuring glucose synthesis in patient's body duringa period of starvation, mark the enzymes and coenzymes;c) list the substrates of this process and its localization.Nonalcoholic fatty liver disease is thought to be exacerbated by a diet heavy in sugar. Explain?10. The patients with a genetically determined medium-chain acyl-CoA dehydrogenase (MCAD) deficiency suffer from episodes of profound fatigue associated with vomiting, which is happening if they fasted for more than 8 hours. They have hypoglycemia, clevated level of free fatty acids in the blood but ketone bodies were below normal. Explain the development of these symptoms. For that answer the question and do the following tasks: a) why hypoglycemia occurs in the patients fasting for more than 8 hours? b) why the level of free fatty acids is elevated in blood? Write the scheme explaining this symptom; c) draw the scheme of fatty acid metabolism, which is blocked in case of this genetic disorder.
- The diabetic body's reliance on ketogenesis to provide energy due to the unavailability of glucose causes serious health and toxicity concerns in these individuals. 1) Describe the role of ketogenesis in diabetes, why the body elects this metabolic pathway, and its resultant benefits and consequences. 2) Describe the role of ketogenesis in a ketogenic diet, why this can be considered a desired metabolic state for individuals, and the benefits and consequences of ketogenesis in these intentional circumstances. Rationalize the differences between these two ketogenic instances.1A. Produceadiagramtoillustratethedifferenttypesofmoleculesfoundincells.Briefly describe their properties and functions within the cell. In a separate diagram, describe how carbohydrates, amino acids and triacylglycerides interact metabolically in the generation of energy in the form of ATP.B. Mineralsandvitaminsareimportantnutritionalmoleculesbecausetheyfunctionas cofactors for critical enzymes in living systems. Without them, important biochemical reactions required for growth and tissue repair will not occur. Knowing this, explain the biochemical significance of folate and iron supplementation in pregnant women and women of reproductive age. Consider the effects of folate and iron deficiency as well. 2A. Thevalueofenzymesinlivingsystemsisthatwithoutthem,chemicalreactionsrequired for life to exist would not be possible under physiological conditions. On the basis of this, discuss how enzymes make life possible from a biochemical perspective. Include the following terms in your…Describe five possible fates for glucose 6-phosphate in the liver.
- n. 5. Calculate the amount of energy released in the liver upon the complete oxidation of a triacylglycerol containing two 6-carbon saturated fatty acids and one oleic acid. Show your calculations. bediosA. List all the materials required for beta-oxidation of fatty acids. How does this process differ from the Biosynthesis of fatty acids? B. Outline the pathway involved in the synthesis of cholesterol from six-carbon intermediate Mevalonate. C. Describe all the stages involved in the urea cycle (Transmination, Ammonia Transport, and stages of the urea cycle).A high-sucrose diet is said to increase the risk of non-alcoholic fatty liver disease. Explain?