The symptoms of Cushing’s disease include abdominal obesity, hypertension, glucose intolerance (steroid diabetes), hirsutism, osteoporosis, polyuria, and polydipsia. Describe the consequences of cortisol excess that would produce each of these symptoms. Which tests can be performed to determine if a patient has primary or secondary Cushing’s disease? What would the results of these tests be for each type of disease?
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The symptoms of Cushing’s disease include abdominal obesity, hypertension, glucose intolerance (steroid diabetes), hirsutism, osteoporosis, polyuria, and polydipsia. Describe the consequences of cortisol excess that would produce each of these symptoms. Which tests can be performed to determine if a patient has primary or secondary Cushing’s disease? What would the results of these tests be for each type of disease?
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- In the initial stages of the disease, type 2 diabetes is treated with Metformin only, and insulin is not needed. In later years, insulin treatment is often needed. Please help explain the progression of the disease, describing the initial role of Metformin as a drug, and the eventual need for insulin.Type- 2 diabetes mellitus is: 1.both a secondary pathology and usually the result of inappropriate hormone secretion 2.both a secondary pathology and usually the result of abnormal target cell responsiveness 3.usually the result of inappropriate hormone secretion 4.a secondary pathology 5.usually the result of abnormal target cell responsivenessIn addition to symptoms of diabetes, all the following are criteria recommended by the American Diabetes Association for use in the diagnosis of Diabetes Mellitus EXCEPT: O Random plasma glucose concentration > 180 mg/dL. O Fasting plasma glucose > 126 mg/dL. O 2-Hour glucose > 200 mg/dL during a 2 hour post prandial glucose test O Hemoglobin A1C > 6.5%
- Identify the cause of each of the following conditions: diabetes insipidus, pituitary dwarfism, gigantism, acromegaly,Cushing syndrome, and Addison disease.Describe the homeostatic control of plasma calcium levels by answering the following two questions: What role does calcitonin play in the maintenance of serum calcium? Give reference to the gland involved in secretion, the bone cells stimulated by calcitonin and the effects of calcitonin. Describe the role parathyroid hormone (PTH) has in the maintenance of serum calcium, giving reference to the gland involved, the particular bone cells stimulated and the major effects of PTH.Discuss the relationship between diabetes mellitus and cardiovascular disease. What dietary measures can be taken to decrease the risks associated with the two disease processes in a patient who has been diagnosed with both?
- Identify the possible etiologies (causes) of Diabetes mellitus. SELECT ALL THAT APPLY A. Reduced effects of insulin on cells of the body B. Increased digestion of carbs in the pancreas C. Pregnancy D. Loss of insulin production by the pancreas.Which of the following describes a feature of the pathophysiology of type 2 diabetes? Question 15 options: a) It is usually an autoimmune disease b) The pancreas makes little or no insulin c) Diabetic ketoacidosis is a common complication d) The liver increases production of glucoseWhat is the most common cause of mortality in those with diabetes mellitus? Question 76 options: a) Renal failure b) Myocardial infarct and stroke c) Gangrene and sepsis d) Autonomic and somatic neuropathy
- Discuss the aetiology and pathogenesis of type 2 diabetes mellitus. In your answer, make clear the term ‘insulin resistance’ by providing an example to illustrate your understanding of this occurrence. Determine whether the C-peptide test would be useful in diagnosing type 2 diabetesWhich of the following best describes the role of the hexosamine pathway in the pathogenesis of the chronic complications of diabetes mellitus? Question 79 options: a) It involves irreversible binding of glucose to proteins, lipids and nucleic acids which damages components of the microcirculation leading to retinopathy, etc. b) It promotes the O-linked glycosylation of proteins and transcription factors, resulting in altered gene expression contributing to insulin resistance & cardiovascular complications c) It promotes the synthesis of DAG which increases pro-inflammatory gene expression and endothelial ET-1 production resulting in blood flow abnormalities d) It leads to intracellular accumulation of osmotically active sorbitol and fructose which damages Schwann cells, erythrocytes and the lens of the eyeWhich of the following statements about type 1 diabetes pathophysiology are true? Select all that apply A) The immune system begins to attack pancreatic B-cells in response to elevated blood sugar B) The activated immune system destroys pancreatic ß cells C) Thelper cells are activated resulting in production of autoantibodies from self-reactive plasma cells D) The body does not produce self-reactive cytotoxic T cells